Preliminary studies reveal conolidine may inhibit specific ion channels, minimizing neuronal excitability and limiting pain alerts. This mechanism is particularly applicable in neuropathic pain, in which abnormal signaling triggers persistent distress. Furthermore, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to pain perception. Computational docking ... https://fordk692axy0.vblogetin.com/profile