Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. On the flip side, our capacity to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK suggests elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by improved pFAK localization https://pro33login11009.idblogmaker.com/31694257/how-pro33-can-save-you-time-stress-and-money